So chronic stress is one of the main contributors to aging. I hope I make it to my 40s.

Do we currently have the ability to do targeted methylation or de methylation at sites in cells?

I suspect that epigenetic aging is adaptive; a deliberately programmed sequence wherein genes more likely to contribute to age-related mortality are shut down progressively. This being in response to random genetic damage accumulating at a predictable rate.

What we think of as aging then becomes primarily the epigenetic response to the problem of DNA damage, much like the symptoms of a virus are mostly the result of the immune response rather than the virus’s direct effects.